Germ-band extension is driven by cell intercalation. (A)
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Forces directing germ-band extension in Drosophila embryos - ScienceDirect
Polarize to elongate
Distinct RhoGEFs activate apical and junctional actomyosin contractility under control of G proteins during epithelial morphogenesis - preLights
Convergent extension requires adhesion-dependent biomechanical integration of cell crawling and junction contraction. - Abstract - Europe PMC
Unipolar distributions of junctional Myosin II identify cell stripe boundaries that drive cell intercalation throughout Drosophila axis extension
intercalation
PDF) There is more than one way to turn a spherical cellular monolayer inside out: type B embryo inversion in Volvox globator
The Canonical JAK/STAT Pathway See text for details.
Jörg GROSSHANS, Professor, Dr. rer. nat Dipl. biochem., Philipps University of Marburg, Marburg an der Lahn, PUM, Faculty of Biology
Short and long germ segmentation: unanswered questions in the evolution of a developmental mode - Liu - 2005 - Evolution & Development - Wiley Online Library
Patterning of the Drosophila Hindgut Epithelium (A) STAT92E protein is
Unipolar distributions of junctional Myosin II identify cell stripe boundaries that drive cell intercalation throughout Drosophila axis extension
Adhesion-regulated junction slippage controls cell intercalation dynamics in an Apposed-Cortex Adhesion Model
Germ-band extension is driven by cell intercalation. (A)
Simulation of clones in a single mutant background. Simulations of
